Trial of William Palmer

Part 27

Chapter 274,102 wordsPublic domain

But suppose for a single moment that excitement of this kind could produce any such effect or influence, where is the excitement manifested by Cook as leading to this supposed disease? They say that the man, when his mare won at Shrewsbury, was full of excitement; and well he might be--his fortunes depended upon the result of that race; and I do not deny for a few minutes he was overpowered by the emotions that the joy of the moment excited in his breast. But that subsided, and we have no further trace of it from that time to the moment of his death. The man passed the rest of the day with his friends in ordinary conversation and in ordinary enjoyment. No trace of emotion was left about him. He is taken ill; he goes to Rugeley; he is taken ill there again; is there the slightest symptom of excitement about the man, or, on the other hand, of depression? Not the slightest in the world. When he is ill, like most other people, he is low; but as soon as he gets a little better he is cheerful and happy; he admits his friends, and he converses with them; the very night of his death, so far from any excitement, his conversation is full of cheerfulness and mirth--he is laughing and happy, little thinking, poor wretch, of the fate that was impending over him. He is cheerful and happy, talking of the future, not in the language of excitement on the one hand, or of depression on the other. What pretence is there for this idle story of excitement and depression? Not the slightest shadow of foundation in the world. But if there were--if those things were capable of producing that form of tetanus which goes by the term “idiopathic,” the character of the disease is so essentially different that it is impossible to mistake the two. What are the cases which they attempt to set up against it? They have brought you the case of Mary Watson, which a gentleman came all the way from Scotland to tell us about. The girl had been ill all day. She was taken with cramps in the night, probably originating in the stomach, extending to all other parts of her body. She gets well in a very short time, and goes about her business. Is that case to be compared for a single instant to the death agony of that wretched man, and the paroxysm that destroyed him? Those are the sort of cases with which they attempt to meet such symptoms as those which are spoken to by the witnesses as accompanying the decease of Mr. Cook.

[Sidenote: Attorney-General]

Gentlemen, I venture, upon the evidence, to assert boldly that the cases of idiopathic tetanus and traumatic, or what I may call natural tetanus, are marked by clear and distinct characteristics, distinguishing them from the tetanus produced by strychnia; and I say that the tetanus which accompanied Mr. Cook’s death is not referable to either of those forms of tetanus. You have upon that point the evidence of witnesses of the highest competency and of the most unquestionable integrity; and upon their evidence I am quite satisfied you can come to no other conclusion but that this was not a case either of idiopathic or of traumatic tetanus. But, then, they say it may have been something else; and various attempts have been made to set up different causes as capable of producing this tetanic disease. And, first, we have the theory of general convulsions; and Mr. Nunneley, having gone through the bead-roll of the supposed infirmities of Mr. Cook, says, “Oh, this may have been a case of general convulsions. I have known general convulsions to assume a tetanic character.” “Well, but pause a moment, Mr. Nunneley, have you ever seen one single case in which death arising from general convulsions, accompanied with tetanic symptoms, has not ended in the unconsciousness of the patient before death!--No, I never knew such a case--not one. But in some book or other, I am told that there is some such case reported”; and he cites, not for that purpose, I think, but he cites, with reference to general convulsions being sometimes accompanied with tetanic symptoms, and ending in death, a very eminent author of the present day. I mean Dr. Copland. Dr. Copland is living, and Dr. Copland might have been called. The author of the book, I apprehend, would stand before you as a higher authority than a man who merely quotes the book as the foundation of his knowledge. Dr. Copland might have been called. Dr. Copland was not called, notwithstanding the challenge which I threw out. Why? Because it is infinitely better in such a case to call together from the east and from the west practitioners of more or less obscurity, instead of bringing to bear upon the subject the light of science which is treasured up in the breasts of the eminent practitioners with whom this great city abounds. Dr. Copland is not called; but I say, as regards general convulsions, the distinction is plain, that where they destroy the patient they destroy consciousness; and here it is unquestionably the fact, that to the last moment of Mr. Cook’s existence, until his burst heart ceased to beat, his consciousness remained.

[Sidenote: Attorney-General]

But then comes another supposed condition from which death in this form may be said to have resulted, and that is the case which was intended to be set up by a very eminent practitioner, I mean Mr. Partridge. It seems that in the post-mortem examination of Mr. Cook, when the spinal marrow was investigated, certain granules were found, and this is seized upon. It is said, “Oh, those granules may have occasioned tetanic convulsions similar to those which were found in Mr. Cook’s case,” and a very eminent gentleman is called to give his opinion upon that subject. I admit him to be not only a man of great eminence, but a man of the highest honour and the most perfect veracity. I allude to Mr. Partridge. I must distinguish between him and other of my learned friend’s witnesses. Some there were who would not be induced, for any consideration in the world, to swerve from what they believed to be the truth. Mr. Partridge is called here to prove that this was a case of what he called arachnitis--inflammation of the arachnoid in consequence of the granules, or some other abnormal condition. I asked him the symptoms which he would find in such a case. I called his attention to what evidently had not been done before, namely, the symptoms of Mr. Cook’s case; and I asked him, in simple, straightforward terms, whether, looking at those symptoms, he would pledge his opinion, in the face of the medical world and the Court, that this was a case of arachnitis, and he candidly admitted that he would not assert that this was in his opinion a case of arachnitis.

Then we have the gentleman who comes all the way from Scotland to inform us, as the next proposition, that Mr. Cook’s was a case of epileptic convulsions with tetanic complications. Now, I asked him this question, “Did you ever know a case of epilepsy, with or without tetanic convulsions, in which consciousness was not destroyed before the patient died?” He said, “No; I cannot say that I ever did, but I have read in some book that such a case has occurred.” “Is there anything to make you think that this was epilepsy?--Well, it may have been epilepsy, because I do not know what else to ascribe it to; but I must admit that epilepsy is characterised generally by a loss of consciousness.” “Well, then, what difference would tetanic complications make?” That he is unable to explain. I remind you of that species of evidence in which the witnesses resorted to the most speculative reasoning, and put forward the barest possibilities without the shadow of a foundation. But this I undertake to assert, and I refer to the evidence to prove it, that there is not a single case either to which they have spoken as coming within their own experience, or of which they have spoken as the result of reading, in which there were the formidable and decisive symptoms of marked tetanus which existed in this case of Mr. Cook.

[Sidenote: Attorney-General]

Having gone through this evidence, I think we have four sets of diseases--general convulsions, arachnitis, epilepsy proper, and epilepsy with tetanic complications. I expected that we had pretty well exhausted the whole of those scientific theories, but we were destined to have another that assumed the formidable name of angina pectoris. I do not know whether it struck you as remarkable that when my learned friend opened this case for the defence he never ventured to assert what would be the nature of the disease to which he would endeavour to refer the symptoms of Mr. Cook; and it must, I think, have struck you as a very remarkable thing that no less than four or five distinct and separate theories are set up by the witnesses who appear on the part of the defence--general convulsions, arachnitis, epilepsy with tetanic complications, and, lastly, angina pectoris. My learned friend had, however, this advantage in not stating to you what was the theory that his medical witnesses would endeavour to set up, because, one after another, I must admit they took me entirely by surprise. The gentleman who was called yesterday at the last moment, and who talked of angina pectoris, would not have escaped quite so easily if I had had the books to which he referred under my hand, and had been able to expose, as I would have done, the ignorance or the presumption of the assertion which he dared to make. I say ignorance or presumption, or, what is worse, an intention to deceive. I assert it in the face of the whole medical profession, and I am satisfied I shall have their verdict in my favour.

[Sidenote: Attorney-General]

But it is a fact which I am entitled to dwell upon, that all those medical witnesses, one and all, differed in the views which they take in this case. There is this remarkable coincidence between the views of some of them and the views of the witnesses whom I called--Mr. Partridge and Dr. Robinson, two of the most respectable witnesses they called, ay, and Dr. Letheby himself, strongly as he was biassed in favour of the defence, being three of the most eminent of the witnesses whom my learned friend brought forward, agreed with the statement made by Sir Benjamin Brodie, and supported by other witnesses whom I brought before you, that in the whole of their experience, in the whole range of their learning and information, they knew of no known disease to which the symptoms of Mr. Cook could possibly be referred. When such men as those tell us such a fact, I cannot but submit to you that it is impossible to exaggerate its importance. But, then, if it be the fact that no known disease can account for such symptoms as those of Mr. Cook, and that they are referable to poison alone, can any one entertain a doubt that that poison was the poison of strychnia? The symptoms, at all events from the time the paroxysm set in, are precisely the same. Distinctions are sought to be made by the subtlety of the witnesses for the defence between some of the antecedent symptoms and some of the appearances after death; but I think I shall show you beyond all possibility of contradiction, that those distinctions are imaginary, and have no foundation in fact. I think I may take this, however, along with me as I go on, that the witnesses called for the defence admit this fact, that from the time the paroxysm set in, of which Mr. Cook died, until the time of the death, the symptoms are precisely similar to the symptoms of tetanus from strychnia. But, then, they say, and this is worthy of your most attentive consideration, that there are points of difference which have led them to the conclusion, or some of them at all events, that those symptoms could not have resulted from strychnia. Let us see what they are. In the first place, they showed that the period which elapsed between the supposed administration of the poison and the first appearance of the symptoms was longer than they have ever observed in animals upon which they have experimented. Now, the first observation which arises there is this, that there is a known difference between animal and human life, in the power with which specific things act upon its organisation, and it may well be that the poison administered to a rabbit will produce its effect in a given time; it by no means follows that it will produce effect in the same time upon an animal of a different description, and still less does it follow that it will exercise its baneful influence in the same time upon a human subject. The whole of the evidence on both sides tends to establish this fact, that not only in individuals of different species, but between individuals of the same species, the same poison and the same dose will produce effects different in degree, different in duration, and different in power. But, again, it is perfectly notorious that the rapidity with which the poison begins to work depends materially upon the mode of its administration. If it is administered as a fluid, it acts with great rapidity; if it is administered in a solid state, its effects come on more slowly; and if it is administered in some indurated substance, it will act with still greater tardiness; and if that substance is difficult of solution, then the period will be still longer before the substance, having become dissolved, is acted upon by the absorbents and taken up into the system.

[Sidenote: Attorney-General]

Now, what was the period at which this poison began to act after its administration, assuming it to have been poison for the purpose of argument? It seems, from Mr. Jones’ statement, that the prisoner came and administered these pills; he saw him administer the pills somewhere about eleven o’clock, so that they were not administered upon his first arrival. The patient, as though with an instinctive sense of the peril which impended, strenuously resisted the attempt to make him take them, and no doubt those remonstrances and the endeavour to overcome them occupied some period of time. The pills at last were taken, and, assuming them to have contained strychnia (which I only do now for the purpose of argument), how soon did they begin to operate? Mr. Jones says he went down after this and had his supper, and came back about twelve o’clock. Upon his return to the room, after a word or two of conversation with Cook, he proceeded to undress and go to bed; and he had not been in bed ten minutes before the warning came that another of these paroxysms was about to take place. The maid-servants put it still earlier; they say that about ten minutes before twelve the first alarm was given, which would make the interval little more than three-quarters of an hour from the taking of the pills and the first manifestation of the symptoms. When, therefore, my learned friend’s witnesses tell us that it took an hour and a half or two hours, we have here another of those exaggerated determinations to see the facts only in the way that will make most for the view which they think proper to put forward. I say it certainly was not more than an hour, and I find in some of the experiments that have been made that the duration of time before which the poison began to work has been little less, if any less, than an hour. Mr. Morley, who is as much entitled to your attention as Mr. Nunneley--indeed, when I contrast the way in which the two men gave their evidence, I am paying him but a poor compliment when I say that he is as worthy of attention as Mr. Nunneley--Mr. Morley says in his evidence that five or six minutes, or something less than an hour, is the period which he observed the poison required to produce its effects upon animals, and in every one of the cases which we have got it will be seen that more than an hour was necessary. In the case of the girl at Glasgow, though I see the medical gentleman speaks of twenty minutes when he was called in, he could have only had that information from the statement of some of the people about. I see the nurse says it was three-quarters of an hour before the pills began to work upon the girl. There may have been some cause for the pills not beginning to take effect within a certain time after their administration; it would be very easy to mix them with some substance that should render them difficult of solution; nay, which might retard their action. I cannot for a single moment bring myself to believe, if in all other respects you are perfectly satisfied that the symptoms, the consequences, and effects were analogous and similar to those produced by strychnia, it is not because those pills may have taken a quarter of an hour or a longer time to manifest their working, it is not on that account you will hesitate to come to the conclusion that strychnia was administered in this case. But then they say, yes, but the premonitory symptoms were wanting here. They tell us in animals they observed that the animal manifests first some uneasiness, shrinks, and gathers itself into itself, as it were, avoids movement, and then certain involuntary twitchings about the head come, those being the premonitory symptoms before the paroxysms set in. They say there were no premonitory symptoms in Cook’s case; I utterly deny that proposition--I say there were premonitory symptoms of the most marked character, though he did not describe them in language. He is lying in bed--he suddenly starts up in an agony of alarm. What made him do that? Was there nothing premonitory, nothing that warned him that the paroxysm was coming? It is clear there must have been. He jumps up in his bed, and says, “Fetch me Palmer, I am going to be ill, as I was last night.” What was it but that he knew the symptoms that attended him on the previous evening were now warning him of what he might expect in a short period, unless succour could be obtained? He sits up, and he prays to have his neck rubbed. What was the feeling about the neck but a premonitory symptom which was to precede the paroxysm which presently supervened? He says, “Rub my neck, it gives me comfort to have it done.”

[Sidenote: Attorney-General]

But here again they take exception, and they say this could not have been tetanus from strychnia, because animals cannot bear to be touched; a touch brings on the paroxysm; not only a touch but a breath of air, a sound, a word, a movement of any one near, will bring on a recurrence of the paroxysm. True; but that is after the paroxysm has once been set up, or when it is just about to begin. It is quite clear that those witnesses who come and say that the fact of Mr. Cook having desired to have his neck rubbed is a fact to prove that this could not be a death from tetanus, have either wilfully suppressed the knowledge in their own minds of the evidence they had heard, or they had paid no attention to it; because in two cases of death from strychnia we have shown the patient endured the touching of the limbs, and found satisfaction from it. In Mrs. Smyth’s case, when her legs were distorted, in the agony of the convulsion she prayed and entreated to have them straightened; she found no additional pain from that operation. The lady at Leeds, in the case which Mr. Nunneley himself attended, implored her husband, between the spasms, to rub her legs and her arms, in order to overcome and subdue their rigidity. That case was under his own knowledge, and, in spite of it, although he detected afterwards strychnia in the body of that unhappy woman, he dares to come forward here and say that the fact of Mr. Cook having before the paroxysm tolerated rubbing, and found comfort in it, proves that this could not have been a death from strychnia. What think you of the honesty of such a witness? But there is a third case, which is the case of Mr. Clutterbuck, spoken to by Mr. Moore. That gentleman had taken an overdose of strychnia, and he suffered from all the pains of tetanus; his only comfort was having his limbs rubbed; and therefore, I say, to contend and to endeavour to persuade a jury that the fact of Cook’s having had his neck rubbed proved that this was not tetanus from strychnia, proves, I say, nothing but the dishonesty and insincerity of the witnesses who can dare to put forward such a pretence.

But, then, they go further, and they say that Mr. Cook was able to swallow. So he was, before the paroxysm came on. Nobody ever pretended that he could swallow after the paroxysm came on. He swallowed the pills, and, what is very curious, and, as they think, bears out and illustrates a part of their theory, is this. It was the act of attempting to swallow the pills--the sort of movement that must have taken place in raising his head and neck for the purpose--that immediately brings on the violent paroxysm of which he died. So far from that in the slightest degree militating against the supposition that this was a case of poisoning by strychnia, it is strongly and decisively conclusive in its favour.

[Sidenote: Attorney-General]

But then they take us to the appearances after death, and they say that there are circumstances to be found which militate against this being a case of strychnia poisoning. Let us see what they are. In the first place, they say the limbs became rigid either at the time of death or immediately after, and that ought not to be found in a case of tetanus from strychnia. Mr. Nunneley says, “In all cases upon which I have experimented I have found the animals become flaccid before death, and they do not become again rigid after it.” I can hardly believe that statement, and I certainly was not a little surprised when the very next witness who got into the box (Mr. Herapath, of Bristol) told us he had made two experiments upon cats, and killed them both. He described them as “indurated and contorted.” Those were his expressions when he found them some hours after death. The presence of rigidity in the body at or immediately after death here is put forward on the part of Mr. Nunneley as one of the grounds upon which he says this was not a death by strychnia, although Dr. Taylor had told us that in the case of one of the cats he killed the rigidity after death was such that upon taking the animal by the hind legs and holding it up in the air, the body maintained its horizontal natural position, as though the animal had been upon its four legs upon a plain surface. Notwithstanding that evidence, Mr. Nunneley had the audacity to say that he did not believe this was a case of poisoning by strychnia, because there had been rigidity of the limbs--because the feet were distorted, the hands clenched, and the muscles rigid as the unhappy man exhibited prior to his death. The very next witness called upon the other side produced two instances in which the animals were indurated from one end of their bodies to the other. As he says they were contorted in all their limbs, and so they remained, it shows what you are to think of the honesty of this sort of evidence, in which facts are selected because they make in favour of the particular hypothesis of the party who brings them forward.

[Sidenote: Attorney-General]